By Ramana S. Moorthy MD

Starts off with an in-depth assessment of immunemedicated eye affliction, summarizing easy immunologic thoughts, ocular immune responses and specified subject matters in ocular immunology. Discusses the scientific method of uveitis and experiences noninfectious (autoimmune) and infectious varieties of uveitis, with an multiplied part on viral uveitis and new fabric on infectious and noninfectious scleritis. more suitable detection of infectious brokers by means of immunologic and genetic tools and new biologic therapeutics are designated. additionally covers endophthalmitis, masquerade syndromes, problems of uveitis and ocular points of AIDS. includes a variety of new colour pictures. significant revision 2011-2012

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Extra resources for 2011-2012 Basic and Clinical Science Course, Section 9: Intraocular Inflammation and Uveitis (Basic & Clinical Science Course)

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Non-B lymphocytes. includes natural killer cells. lymphokine-activated cells. and ki ller cells. Antibodies. or immunoglobulins. are soluble antigen-specific effector molecules of adaptive immunity. After appropriate antigenic stimulation with T-Iymphocyte help. B lymphocytes secrete IgM antibodies and. later. other isotypes. into the efferent lymph fluid draining into the venous circulation. Antibodies the n mediate a variety of immune effector activities by combining with antigen in the blood or in tissues.

During the processing phase of the primary response. 000) and then stimulate these cells from a completely resting and naive state. a sequence that requires days. The secondary processing response for T and B lymphocytes is shorter for at least 3 reasons: • Upon removal of antigen. T and B lymphocytes activated during the primary response may gradually return to a resting state. but they retain the capacity to become reactivated within 12-24 hours of antigen exposure. They are now memory cells rather than naive cells.

Including most tumor peptides or viral peptides after host cell invasion. MHC class II molecules (ie. HLA-DR. -DP. and -DQ) serve as the antigen-presenting platform for CD4. or helper. T lymphocytes (Fig 2-3). All APCs for CD4 T lymphocytes must express the MHC class II molecule. and the antigen receptor on the helper T lymphocyte can recognize peptide antigens only if they are presented with class II molecules Simultaneously. However. only certain cell types express MHC class II molecules on their plasma membrane.

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