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Read Online or Download 2008-2009 Basic and Clinical Science Course: Section 5: Neuro-Ophthalmology (Basic and Clinical Science Course 2008-2009) PDF

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Additional info for 2008-2009 Basic and Clinical Science Course: Section 5: Neuro-Ophthalmology (Basic and Clinical Science Course 2008-2009)

Sample text

The macular fibers tend to move superolaterally. Cortical and subcortical pathways may modulate activity in the lateral geniculate. Pathways have been identified from the raphe nucleus and the locus ceruleus. In addition, the cortex, superior colliculus, and pretectal nuclei project back to the lateral geniculate. Cortex Following a synapse in the lateral geniculate, the axons travel posteriorly as the optic radiations to terminate in the primary visual (calcarine) cortex in the occipital lobe (Fig 1-13).

Burst cell input may come in part from the PPRF caudally but also locally within the riMLF. The INC receives signals from the riMLF and from the vestibular nuclei and projects to the motoneurons of the third and fourth nerve nuclei through the posterior commissure. Activity from the vertical gaze center is distributed to the oculomotor (CN III) and trochlear (CN IV) nuclei. Information involved in upgaze crosses in the posterior commissure. Damage to this pathway in the dorsal midbrain results in Parinaud dorsal midbrain syndrome, which clinically includes vertical gaze difficulty (most commonly impaired supraduction), skew deviation, light-near pupillary dissociation, lid retraction, and convergence-retraction nystagmus (CRN).

The INC receives signals from the riMLF and from the vestibular nuclei and projects to the motoneurons of the third and fourth nerve nuclei through the posterior commissure. Activity from the vertical gaze center is distributed to the oculomotor (CN III) and trochlear (CN IV) nuclei. Information involved in upgaze crosses in the posterior commissure. Damage to this pathway in the dorsal midbrain results in Parinaud dorsal midbrain syndrome, which clinically includes vertical gaze difficulty (most commonly impaired supraduction), skew deviation, light-near pupillary dissociation, lid retraction, and convergence-retraction nystagmus (CRN).

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